Pre-analytical Considerations
- CSF must be collected via lumbar puncture under standardized conditions.
- Collect at least 1–2 mL in polypropylene tubes, ideally from the L3/L4 or L4/L5 space, avoiding traumatic tap.
- Freeze immediately at –80°C if not analysed promptly.
- Paired blood samples are often helpful.
Key CSF Neurotransmitter Metabolites & Normal Values
Metabolite | Normal Range (Approximate)* | Clinical Significance |
5-HIAA (5-Hydroxyindoleacetic Acid) |
40–120 nmol/L |
Major serotonin metabolite. ↓ in AADC deficiency, serotonin synthesis defects, or MAO-A deficiency. |
HVA (Homovanillic Acid) |
150–450 nmol/L |
Major dopamine metabolite. ↓ in AADC deficiency, tyrosine hydroxylase deficiency, ↑ in MAO-A deficiency, dopamine transporter defects. |
HVA:5-HIAA Ratio |
1.0–4.0 |
A high ratio suggests MAO-A deficiency or dopamine transporter defects. A low ratio may occur in serotonin-specific disorders. |
MHPG (3-Methoxy-4-hydroxyphenylethyleneglycol) |
10–50 nmol/L |
Norepinephrine metabolite. ↓ in dopamine β-hydroxylase deficiency. |
3-OMD (3-O-Methyldopa) |
<10 nmol/L |
↑ in AADC deficiency. Marker of excess L-DOPA methylation. |
L-DOPA |
<10 nmol/L |
Precursor to dopamine. Elevated in AADC deficiency, aromatic amino acid decarboxylase defect. |
5-HTP (5-Hydroxytryptophan) |
<5 nmol/L |
↑ in AADC deficiency, ↓ in tryptophan hydroxylase deficiency. |
Neopterin |
30–70 nmol/L |
Marker of immune activation. Very ↑ in AADC deficiency. May help differentiate primary neurotransmitter defects. |
Biopterin |
10–30 nmol/L |
Co-factor for tyrosine and tryptophan hydroxylase. Abnormal in BH4 metabolism disorders. ↓ in GTPCH, PTPS, and DHPR deficiency. |
Pterin Profile (Neopterin/Biopterin ratio) |
N/A |
Characteristic patterns in BH4 pathway disorders. |
*Ranges can vary between laboratories; consult reference lab-specific cut-offs.
Clinical Interpretation: Common Patterns
Disorder | CSF HVA | CSF 5-HIAA | 3-OMD | Biopterin/Neopterin | Notes |
AADC deficiency |
↓↓ |
↓↓ |
↑↑ |
Normal or ↑ neopterin |
Severe hypotonia, oculogyric crises, dystonia |
Tyrosine hydroxylase deficiency |
↓↓ |
Normal |
Normal |
Normal |
Infantile parkinsonism, dopa-responsive dystonia |
GTPCH deficiency |
↓↓ |
↓↓ |
Normal |
↓ biopterin |
Dopa-responsive dystonia, treatable with BH4 |
MAO-A deficiency |
↑ HVA & 5-HIAA |
↑↑ |
Normal |
Normal |
Aggression, developmental delay, ↑ HVA:5-HIAA ratio |
Dopamine transporter defect (DAT) |
↑ HVA |
Normal |
Normal |
Normal |
Paroxysmal dystonia, oculogyric crises |
DHPR deficiency |
↓ HVA & 5-HIAA |
↓↓ |
↑ phenylalanine |
↓ biopterin |
May present like PKU, also affects neurotransmitters |
When to Suspect a Neurotransmitter Disorder
- Early-onset movement disorders (dystonia, parkinsonism, chorea).
- Recurrent oculogyric crises.
- Autonomic dysfunction (hypersalivation, temperature instability).
- Treatment-refractory epilepsy.
- Profound hypotonia with preserved cognition.
- Unexplained developmental regression.
Relevant Tests to Combine
- Plasma amino acids.
- Serum prolactin (elevated in dopamine deficiency).
- Urine purines/pyrimidines.
- Genetic testing (targeted panels or WES).